Series-1Sep. 2020 Issue Statistics
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Abstract: Aim: To assess the clinical and functional results of using a Dynamic hip screw in the treatment of stable intertrochanteric fractures. Methods and materials: Over a period of one year starting from November 2016 to November 2017, 20 patients diagnosed with stable intertrochanteric fracture classified as Type 1 Boyd and Griffin classification were included in the study. All cases were treated with closed reduction and internal fixation with a dynamic hip screw by a single surgeon. Harris Hip score was used to assess functional outcome. Results: We had 11 male and....
Key Words:Intertrochanteric fractures, proximal femoral fractures, dynamic hip screw, DHS
[1]. Senohradski K, Markovic-Denic L, Lesic A, Bumbasirevic V, Bumbasirevic M. Trends in the incidence of hip fractures. Osteoporos Int. 2013; 24:1759-63. Doi:10.1007/s00198-012-2244-y.
[2]. Ngobeni RS. Mortality in elderly patients with intertrochanteric fractures: three years' experience. SA Orthop J. 2010; 9:55-60.
[3]. Braithwaite RS, Col NF, Wong JB. Estimating hip fracture morbidity, mortality and costs. J Am Geriatr Soc. 2003; 51:364-70.
[4]. Magaziner J, Fredman L, Hawkes W, Hebel JR, Zimmerman S, Orwig DL et al. Changes in functional status attributable to hip fracture: a comparison of hip fracture patients to community- dwelling aged. Am J Epidemiol. 2003; 157:1023-31. DOI: 10.1093/ aje/kwg081.
[5]. Hallberg I, Bachrach-Lindstro ̈m M, Hammerby S, Toss G, Ek AC. Health-related quality of life after vertebral or hip fracture: a seven-year follow-up study. BMC Musculoskelet Disord. 2009; 10:135. DOI: 10.1186/1471-2474-10-135.
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Abstract: Objective(S): To compare the incidence of placenta previa, associated factors, complications, placental position, mode of delivery and fetal and maternal outcome in non-scarred (Group A) and scarred uterus(Group B) over 24 months of a hospital based study. Materials and methods: In a prospective study, 112 cases of pregnancies beyond 28 weeks of gestation, complicated by placenta previa were identified. These cases were divided into two groups, non-scarred(Group A, n=33) and scarred uterus( Group B, n=79). Out of total 13532 deliveries at the hospital over study period, 6898 women had history of previous 1 or more LSCS or 2 or more curettage and total of 6634 women had nonscarred uterus.....
Key Words: Feto-maternal outcome, Placenta previa, Scarred uterus
[1]. ACOG committee on obstetric practice. ACOG committee opinion number 266, Jan 2002 : Placenta accrete obstet Gynecol 2002 99: 169-70
[2]. Awan N, Bennett MJ, Walters WA: Emergency peripartum hysterectomy: a 10 year old review at the Royal Hospital for Women, Sydney. Aust N Z J obstet Gynaecol 51(3):210, 2011
[3]. Benirschke K, Burton, Baergen RN: Pathology of the human placenta, 6th ed. New York, Springer, 2012, p 204
[4]. Bencaiova G1, Burkhardt T, Beinder Abnormal placental invasion experience at 1 center E J Reprod Med. 2007 Aug;52(8):709-14
[5]. Biro MA, Davey MA, Carolan M, et al: Advanced maternal age and obstetric morbidity for women giving birth in Victoria, Australia: a population-based study. Aust N Z J Obstet Gynaecol 52(3):229, 2012
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Abstract: Objective- The study was done to see the prevalence of cholelithiasis in chronic kidney disease(CKD) patients and establish CKD as a risk factor for cholelithiasis. Materials and method- Medical, demographic and clinical data of all the patients were recorded between age of 18-70 years with diagnosis of CKD as per NFK/KDOQI criteria regardless of its primary cause.The serum creatinine and blood urea was estimated at the time of admission and eGFR was calculated. USG abdomen was done during hospital stay.
Results- Overall prevalence of cholelithiasis in CKD patients was 18.9%. Prevalence rate of cholelithiasis was increasing with age and body mass index(BMI), much higher in female, dyslipidemic, diabetic and patients with secondary hyperparathyroidism...
Keywords: CKD, Cholelithiasis, BMI, eGFR.
[1]. J.Larry Jameson et al,2016;Harrison's Principles of Internal medicine19:1811-1825
[2]. Singh A, Farag Y, Mittal B, Karai Subramanian K, Reddy SRK, Acharya VN.et al,2003; Epidemiology and risk factors of chronic kidney disease in India-result from the SEEK (Screening and Early Evaluation of Kidney Disease) study. BMC Nephrology 14:114-23
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[5]. Campese VM, Romoff MS, Levitan D, Lane K, Massry SG,1981;Mechanisms of autonomic nervous system dysfunction in uremia. Kidney Int. 20:246Y253.
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| Paper Type | : | Research Paper |
| Title | : | Acute visual dysfunction following Phenytoin induced Toxicity |
| Country | : | India |
| Authors | : | Dr. Shailendra Batham || Dr. Jitendra Kumar |
| : | 10.9790/0853-1909011517 ![]() |
Abstract: Aim: Acute Visual Dysfunction may be caused by Acute Phenytoin Toxicity Method: An 18 year old female with prior generalized tonic-clonic seizures developed blurred vision, diffuse corneal opacity (OD), and ankyloblepharon after Phenytoin administration for seizures. Colour vision was found to be normal with Ishihara pseudo-isochromatic charts and visual fields (Humphrey's automated perimeter) show gross concentric constriction in both eyes. Fundus examination revealed increased CD ratio in both eyes. Patient also developed multiple cutaneous maculo-papular lesions and Steven-Johnson syndrome like exfoliation of skin around lips and perioral area. Serum free-phenytoin concentration measured reveals toxic levels of Phenytoin with no other prior co-morbid retinopathy or optic nerve defect. Results: Phenytoin was withheld, and Leviteracetam.....
Keywords: acute phenytoin toxicity, ocular manifestation, Symblepharon, SJS-Steven Johnson Syndrome
[1]. Shaikh AS, Li Y, Cao L, Guo R. Analysis of phenytoin drug concentration for evaluation of clinical response, uncontrolled seizures and toxicity. Pak J Pharm Sci. 2018 Jul;31(4(Special)):1697-1700. [PubMed]
[2]. Manto M, Perrotta G. Toxic-induced cerebellar syndrome: from the fetal period to the elderly. Handb Clin Neurol. 2018;155:333-352. [PubMed]
[3]. Sasaki E, Yokoi T. Role of cytochrome P450-mediated metabolism and involvement of reactive metabolite formations on antiepileptic drug-induced liver injuries. J Toxicol Sci. 2018;43(2):75-87. [PubMed]
[4]. Gosavi DD, Akanksha S, Sanjay N. A case of phenytoin induced gum enlargement. Asian J Pharm Clin Res. 2012; 5(1): 10-1
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Abstract: Reactive perforating collagenosis ( RPC ) is characterized by transepidermal elimination of altered collagen through the epidermis. Two distinct forms of RPC are known ; rare inherited form that starts in early childhood and a common acquired form that occurs in adulthood. We report a rare case of inherited form of RPC in a female child with presence of similar lesions in her elder brother .A 9 year old female child presented with extensive dark colored skin lesions over face and extremities since 6 months. On dermatological examination , multiple closely studded skin colored and hyperpigmented crateriform lesions with central crusting were present on dorsum of hand. Discretely arranged papules with central adherent keratotic plugging were seen over trunk and extremities. Histopathological examination revealed invagination of the epidermis with necrotic inflammatory material and collagen fibres which are seen at the base of the lesion with transepidermal elimination . Masson's......
[1]. Verma R, Vasudevan B, Pragasam V, Deb P, Venugopal R, Mitra D. A rare case of familial reactive perforating collagenosis. Indian J Dermatol 2013;58:408
[2]. Kandhari R, Sharma V, Ramesh V, Singh A. Familial reactive perforating collagenosis in three siblings. Indian J Dermatol Venereol Leprol 2014;80:86-7
[3]. Kumar V, Mehndiratta V, Sharma RC, Narayan S, Koranne RV, Kakar N. Familial reactive perforating collagenosis: A case report. J Dermatol 1998;25:54-6.
[4]. Bhat YJ, Manzoor S, Qayoom S, Wani R, Baba AN, Bhat AH. Familial reactive perforating collagenosis. Indian J Dermatol 2009;54:334-7.
